to generation, that is, just a separate gene, that is, you and i don’t have such a gene, pah-pah-pah gene, or rather, we are not talking about a gene, about some kind of polymorphism, yes, that is, about a gene variant, some kind of amino acid replacement that works a little not so, that is, it is a damaged version of the gene. at healthy people also have this gene, but in people with a predisposition or patients, it is slightly changed, and you know what this change is, you generally need to know, if a patient comes to you, what is the cause of his genetic disease? yes, of course, the answer is yes and yes, because, firstly, there are breakdowns that greatly affect the behavior of the tumor, yes, there are mutations that make the tumor insensitive to conventional therapy, well, here is the simplest example, very well known in oncology gene, not only in pediatric oncology, but an adult has such a gene p-53, yes, which encodes a protein that controls cell death and the cell’s response to genetic damage, but if such a damaged version of such a gene is inherited, then - firstly, the pat

gene.ff that is the late eighties where you're t■ealking where the competition starts heating up. you have the tools for the first time to do sequencing. that is, you have the tools kind of look inside individual genes and see where occur. and so there were two teams unbeknownst to each other, that had■lead from any serum. these cancer ridden families trying to find what is is there a specic specific gene that c in all the family members who haveembers who don't have cancer, they don't have that mutation. ere ara lo&3of genes in the body. so kind of figuring out where to look■hat they have. and so two teams, both starting in 1989,ou this gene, seek relatively new sequencing ch which very laborious. nowadays can be done, yoowe tecy improves it'll be done very fast. but it was very laborious back then amazingly, after more than a year of looking at the genes and doing all this gene in august 1998, both identify a mu gene p53 after looking at other genes at is in common in all these different

the status of a gene? very large breakdown, yes, a chromosome break, the exchange of large pieces of chromosomes among themselves, or maybe a very small breakdown, like the replacement of one letter among the billions that actually make up the genome of the cell, but from this point of view, any oncological the disease is genetic in nature and i still remember it. sometimes there are some phrases that you don’t need to read the whole book, sometimes it’s enough to read the title, so i still have in my head such a phrase from the book that you should be more familiar with was such a book by alberts, molecular biology cells, yes, this is such a university bible, ours is molecular, and at the very end there was a subchapter dedicated to cancer onology. in a sense this is, that is, the cells of our body, and cells of our body, somatic genetic disease of somatic cells, cells, this means those cells that will not pass on their genetic information to offspring, there are germinal cells, cells that are either game

gene. so you have all this stuff that is evolving. finally, you get to the late eighties where you're talking where the competition starts heating up. you have the tools for the first time to do sequencing. that is, you have the tools kind of look inside individual genes and see where occur. and so there were two teams unbeknownst to each other, that had tissues from families with lead from any serum. these cancer ridden families that are both trying to find what is is there a specific mutation in a specific gene that is common in all the family members who have cancer. but for the family members who don't have cancer, they don't have that mutation. there are a lot of genes in the body. so kind of figuring out where to look is one of the big challenges that they have. and so two teams, both starting in 1989, you kind of start this search using this gene, seek relatively new sequencing technology, which very laborious. nowadays can be done, you know, kind of because technology improves it'll

, that is, it is a damaged version of the gene; healthy people also have such a gene, but people withu generally need to know, if a patient comes to you, what is the cause of his genetic disease? yes, of course, the answer is yes and yes, because, firstly, there are breakdowns that greatly affect the behavior of the tumor, yes, there are mutations that make the tumor insensitive to conventional therapy, well, here is the simplest example of a gene that is very well known in oncology, not only in pediatric oncology, but also in adults. the p-53 gene, yes, which encodes a protein that controls cell death and the cell’s response to genetic damage, but if such a damaged version of such a gene is inherited, then - firstly, the patient has a much greater risk of developing various tumors, including also because in this case , cell death is good, that is, if this gene is damaged, then the cells do not die. she's dying does not know how to die when needed, yes, that is, she does not obey instructions and does not die, for example, patients with mutations in such a gene, for example, do not re

and it turns out this gene is very important because it's a cancer gene.onstantly dividing and most of the divisions, you know, the mutations that we have when our cells are dividing are benign. occasionally there is mutation that could become malignant, it could be cancerous. but amazing gene that over millennia has evolved into what is called a cancer suppressor gene. it's a it's ■t seek and destroy gene. it fine is potentially cancer cells, identifies them and either kills them or i mean, how amazing is that? the problem, of course, if you're born with gene in a b■s nearly as well.esn't do its but this was a huge, huge know, kind of raised, you know, kind of a lot of a lot of excitement. but it also explained a lot like, for example, you know, my family,a why there were so many cancers. so it turns out, you know, kind of if you'res inherited mutation, the figures ared, you know, kind of heartbreaking. by age 40, inherited mutation, there's a 50% chance that you'll have ve had cancer. often multiple cancers, often starting at early ages. it's a versus a

gene.t to tat talking where the competition starts heating up. you have t for the first time to do sequencing. ave the tools kind of look inside individual genes and see where and so there were two teams unbeknownst to eachm< oth tissuh lead from any serum. families that are both trying to find is there a specific mutation in a spefic gene that is common in all the family mbers who have cancer. but for the family members who don't have cancer,hey don't have that mutation. there are a lot of genes in the body. so kind of figuring out where to look is one of the big challenges that theyand so two s search using this gene, seekrelg technology, which very laborious. nowadays can be done, you know, kind of because technology fast.s but it was very laborious back then amazingly, after more than a year of looking at the genes and doing all this gene in august 1998, bothlç identify a mutation on the same gene p53 after looking at other genes that is in common in all these di families and and neithe

gene therapy aims to address the underlying causes of disease, gene mutations which as you know our mistakes in the basic blueprints of our body. gene therapy can fill in missing parts or correct errors in those blueprints. early in my career doctors like me had few therapeutic options to offer these patients. not being satisfied with this, 1995 i said the first team of researchers to use aav as a vector or carrier in gene therapy trial for cystic fibrosis. aav vectors one of the basic building blocks of today's gene therapy products. vectors with the genetic information they taken directly target the cause of disease and change the way a cell functions. gene therapies only to be administered one time to have long-lasting potentially curative effect. but of the thousands of rare diseases, only handful are being addressed currently with the products that are already licensed, and a small subset of the total number are being addressed with drugs in the pipeline. the question now is how can we accelerate the path from gene to therapy and make these challenges featured over, , when there is a li

gen seek and destroy gene.cells, identifies them and either kills them orem. i mean, how amazing is that? the problem, of course, if you're born with gene in a mutated form,do its job nearly as well. but this was a huge, huge breakthrou that that, you know, kind of raised, you know, kind of a lot of a lot of excitement. but it also explained a lot like, for example, you know, my family, family a why there were so many cancers. so it turns out, you know,2u kid with this inherited mutation, the figures areering and, you know, kind of heartbreaking. by age0, if have this inherited mutation, there'shat'e had cancer. often multiple cancers, often starngit's a versus a 5% chancee overall population. andall the way out. that's how pernicious this 's not a question of if, it's a question of many. yeah. and if there's a 5050 chance, you know, that you'll inherit it from one from the parent that has. yes, yes. there is a 5050 chance.urns out, which was know ki of, y quirk fd of the luck of the scientific, scientific and

gene. the relevant gene from each parent. it's not enough to inherit the copy from one parent. so what happens in this community is, is that because it's reproductively isolate the gene for deafness spreads broadly within the population ultimately numerous deaf individuals may be born into community and then a sign language may emerge here in communities and this is important may also know and use the sign language in fact the last users village sign language is at least a last users in. the village are often hearing individuals. so why should we be interested in village sign languages? well, they are very instances in which we can actually observe. they emergence of a new language. it's almost impossible to find new amongst the spoken languages, but it is possible to identify new sign languages. new languages to new sign languages that have no prior that are not derived from any previously existing sign language. and in fact some modern village sign languages only a few generations. so thi

and today this forty is an indispensable gene source for the creation of new high-yielding varieties can see the furniture interiors, everything that nikolai dmitrievich mukhin had in his office, stationery supplies, work notes, everything he had to work with. for every day. the pride of our museum is a truly valuable award for our institution, this is the order of the red banner of labor. we were awarded these awards in 1977, on the fiftieth anniversary of our founding, for the development of agricultural science and the creation of new varieties of agricultural plants. dear colleagues, today there will be a meeting of the council of young scientists on the results of scientific activities. the first quarter of 2024 is undoubtedly in the center of in agriculture, we are working with young specialists, we are making an application for internship for students of agronomic faculties in such a way that in the future we can accept them into graduate school, give them relevant work so that they can...

bank, in general more than 53,000 collection samples, unique samples, because the world gene pool, them fund of our belarusian scientists, which is the basis of our collection, we can use it for exchange with other countries, and of course, we transfer everything to our breeders, conditions have been created here, three types of conservation of plant resources have been created: long-term, medium-term, and short-term seed storage. we are in the chamber where the active collection of our genebank is stored. the eggs are stored at a temperature of 0 +3°. this collection is intended for exchange and active use by our breeders. the collectible image is saved on for 15 years, does not require reseeding and a collection of about 15,000 collection specimens is stored in this chamber in this storage. samples are registered. encoded under a certain number, they are entered into the database, where each sample has its own place, its own rack, its own shelf, so it is easy and simple for every employee of our gene bank to find this or that sample, this material is the basis for breeding activities

, and olya, what is a gene pool, an agricultural gene pool, what does it all look like, and this is abase of seeds, varieties, samples that scientists can use for their own purposes, mainly, of course, for selection, so that an idea that, say, a scientist has somewhere in his head, is realized. breeders can be called real artists, because they improve and develop completely new plant varieties. it works and is not easy, but terribly interesting. in simple terms, i’ll ask, if a global catastrophe occurs in the world, then as i understand it, it is the bank, this gene pool, the agricultural gene pool that will save all of humanity, save breeding, ensure food security at the level of, say, the state, that will be preserved here. reserve for the creation of new source material, seeds that already exist and provide a supply of new varieties. last question, when will bananas start growing in belarus? they are in the botanical garden of the academy. so, you imagine, everything is here. and it's not empty sound. i wanted to drink tea, but olga had everything ready for this. i believe that her

olya, what is the agricultural gene pool? what does it all look like?torage, a database of seeds, varieties, samples that scientists can use for their own. mainly, of course, for selection, so that an idea that, say, a scientist has, somewhere in his head, is implemented. breeders can be called real artists, because they improve and develop completely new plant varieties. the work is not easy, but terribly interesting. i’m asking in simple terms, if a global catastrophe happens in the world, then as i understand it, it’s the bank, this gene pool, the agricultural gene pool. will save all of humanity, will save selection, will ensure food security at the level of, say, the state, here the seed base is preserved, which already exists and provides a reserve, a reserve for the creation of new source material, new varieties. last question: when will bananas start growing in belarus? are they in the academy's botanical garden? so, you imagine, everything is here. and this is not an empty phrase. i wanted to drink tea, but olga had everything ready for this. i

provides for the storage of a collection sample for up to 40 years, this is the golden fund of our gene the basic collection is preserved, this is ... vacuum packaging with seeds that are stored for 40 years, you you see the national number, which carries certain information, and in accordance with this number we can also determine where the collection specimen is located, where it is located in the gene bank, and this collection is always maintained in a living form, this guarantees food. the security of our country, the guarantee of this food security, undoubtedly over the long period of operation of the institute, about the subsequent scientific and practical center, there was an accumulation of material, this is the improvement of production tools, this is ... the development of methods, these are the signs of attention to scientists. we are in the history museum of the scientific and practical center of the national academy of sciences of belarus for agriculture. the opening of our museum took place in 2013 on the eve of the day of belarusian science. this project collected a large

are you living the gene? absolutely. it is| the gene? absolutely. it is absolutely _ the gene?cinating. -- l absolutely fascinating. —— living the dream. thank you for joining us. the tennis legend roger federer has attended a screening of his upcoming documentary, at the tribeca festival, in new york. the film which is called, federer — 12 final days, follows the 20—time grand slam winner during the last 12 days of his career before his retirement. he said it was originallyjust meant to be for his family and friends but developed into the documentary. the film has its premiere in london on thursday. well, i think that is the beauty of this film as well, it shows you can be brutal rivals, tough competitors but then you can come out and get along and have so much respect for one another. it is a good message to the well, to the kids, all the competitors like juniors who take life so serious that when it comes to their matches, the aircraft and when they see that me and wrap nadal, yelling —— novak djokovic get along. coming up ahead on business today, do you use that fitness app

it also says how legal it is. bison from the main gene pool to the reserve one.on, friendly countries buy our bison to breed them in their reserves. of course, healthy, young individuals are sold for sale. in may, the first batch of nine heads went to bashkartastan; before that, zubrov bought kazakhstan from us. that is, it has long been legal to sell and shoot bison, because the optimal number for preserving the population in our country is 1,500 individuals, and we now have 2.0 of them. 616, this does not mean that you can distribute them and shoot from right to left, but there is no reason to be afraid of the imminent disappearance of this species. by the way, belarus is a country with exciting modern history. our bison is a rare animal, and i think it’s right to take the best from the experience of friends. therefore, in the future , we could conduct bison diplomacy, like china does with pandas, leasing them only to states with which it is ready to do business. we could build the same scheme with the sale of teeth, only to friendly countries. how many individ

plant genetic resources, collection specimens are being studied and seeds are concentrated in our gene bank, in general more than 53,000 collection specimens samples, unique samples, because the world gene pool, the gene pool of our belarusian scientists, is collected here, which is the basis. type of conservation of plant resources is long-term, medium-term and short-term seed storage. we are in a chamber where the active collection of our gentbank is stored, collection samples are stored at temperature, but... this collection is intended for exchange and for the active use of our breeders. the collection pest persists for 15 years, does not require reseeding and a collection of about 15 thousand collectible specimens is in this in this chamber in this storage. samples are registered under a specific number and coded. played by the western selection center for grains, leguminous cereal crops, it was organized in 1970, this period is characterized by the intensification of the selection and senovic process, from the creation of the institute in 1927 until the creation of the western se

gene, thank you. always good to see you. >> thank you. >> gene munster., i want to go back to a comment you made, microsoft has to work with apple. do they? or can they own the enterprise and apple owns the user? >> okay, that's fair, except for, you know, i think that's kind of the world we live in today, sntisn't it? this is a case where chatgpt seems to be more than an enterprise dynamic. and bringing it to the consumer -- until proven otherwise, apple is the conduit. they are the distribution channel for every one of these major tech companies. that includes meta, who a lot of people are doing -- how much interaction are people doing on a pc? my expectation is that we still have to be using apple products and today didn't change that. >>> meantime, nvidia wrapping up its first trading day post split. shares closed up three-quarters of a percent to just over $121 after the company's split took effect, but not all semis were in the green. amd dropping 4.5% after a downgrade at morgan stanley. analysts cutting its rating to equal weight, saying the a.i.

the flagship collection of the belarusian plant gene pool bank is kept by its walls of our academy ofscientific and practical center for agriculture; its headquarters are located in zhodino. there is a room with complex equipment, brake controls, and almost 47 collectible plant specimens. they are like advanced. storages of the world they sleep in aluminum envelopes and jars, packaged in small portions, including the best foreign and purely belarusian varieties, there are those that have no analogues at all in the world, for example, pygmy wheat, it was recognized as unique and was taken to that same planetary storage facility at spitsbergen. this collection exists because alexander lukashenko did not allow the soviet school of seed production to be destroyed. the president instructed the country's biologists and geneticists to develop a program for preserving the gene pool of plants, right now. the result of this program is recognized as a national treasure, based on the samples, the collection was filled with belarusian seeds of native domestic selection. the share in some cultures

a gene, pah-pah-pah, but someone has it, maybe you and i have such a gene, or rather, it’s more likely much more the risk of developing various tumors , including because in this case cell death is good, that is, if it is... the gene is damaged, then the cells do not die, the cell dies poorly, it does not know how to die when it is needed, yes, then there is she does not obey instructions and does not die, and here are patients with mutations in such a gene, for example, they respond poorly to conventional treatment, and they require special treatment, including bone marrow transplantation, and another reason why it is important for us to know this genetic predisposition is because then we can counsel the family how are you? they cannot be removed surgically, they are most often immediately, well, in a sense, we can say that this is the fourth stage, yes, if we speak in simple language, that is, they are distributed throughout the body, so that they to cure surgery is not just not enough, surgery doesn’t work here, and drug therapy is needed, immunotherapy methods are now appearing, th

let's bring in cpa and president of the marx group, gene markets so, gene -- marx.lways good to have you. i know you sent in some notes, some suggestions. let's go down those. >> sure. arthel: you said to maximize interest income. so how do you do that? >> yeah, arthel, first of all, interest expense is high, but there are opportunities by maximizing interest income. you just mentioned that inflation right now is around 3%, but most banks are offering cds that pay, certificates of toes if sit, that pay anywhere from 5-6% in interest. or if you don't want a cd, you want a treasury note or a treasury bill or talk to an insurance company about a multiyear annuity, all of these things are paying in excess of 5%. the fee you have to be careful though, when you put money into now these vehicles, you are locking them up for a period of time. it might be six months or a year or even longer. so you are if you've got excess cash that you don't think you're going to be using, put it away in a cd. you'll earn almost twice what inflation is now. it's a good way to take advantage

very rapidly, in the development of the immune system there is such a very important stage when the genes encoding antibodies are rebuilt very intensively, that is, literally at the dna level, cross-linking cuts occur in order to form such a lot. antibodies, we need millions, there may be hundreds of millions of antibody specificities to the immune system could protect us from any infections, precisely in these first years of life, the intensity of the formation of cells of the immune system, lymphocytes, these are billions of cell divisions there every day, yes, and with each division these small breaks in the dna cross-link occur. sometimes, apparently, very rarely, in fact, an error occurs there, if as a result of such a break the gene that controls cell division or affects the death of the cell is damaged, then there will be trouble, yes, then such a cell can turn into a tumor, well in a practical sense, considering that this is, well , obvious, that this is a tragedy for the family, yes, that is, that is, there are some measures that can be taken at a certain age of the child to redu

gene said, i'm not going to take it back. you clearly still disagree. >> i still disagree, despite an 8% rise. gene is a great analyst, covering this for a very long time. and at the end of the day, apple may have had its biggest market cap gain in its history too on the back of this. i'm just hard pressed to believe that much of what they reported yesterday or detailed is going to be usable in the next iteration of the iphone that comes out in september and october. if you can justify the sort of move that the stock had today, you really have to be able to think that earnings are going to go up meaningfully, that from a midsingle digit sort of thing and revenues in midsingle digits, i'm not sure about that. again, i think that investors are getting ahead of the street, meaning wall street analysts, i did not see a bunch of analysts come up and upgrade the stock or raise their calendar 2025 earnings. so, again, you know, we are in a market where there seems to be no shortage of enthusiasm for stories that are perceived to wor

chromosome level is called 92, the ninth and twenty-second chromosomes exchange pieces, a chimeric genes formed, the so -called bcr able, that is, a gene that pathologically connects two ordinary proteins they begin to give to the cell a signal for us to share well and die badly, that is, is this a philadelphia problem or do we have it too? with the help of one or more medications depends on the type of leukemia, well, except for the actual chromosomal rearrangements, which are simple, well, in a certain sense, simple to look at, but this does not exhaust all the diversity, hematological diseases of children, there are a lot of so-called single nucleotide polymorphisms, or, well, if you want point mutations, where one letter or several letters are replaced, that’s also yes several nucleotides, that is, letters. here in this genetic code, and such a replacement can be no less effective in turning a cell into a tumor one, but it is impossible to notice it at the level of an entire chromosome, and here and what do you do then, and here you need methods of so-called dna sequencing, yes, tha

and we speak to one man with the bleeding disorder haemophilia who says gene therapy has made him feel cured. and coming up on bbc news... having beaten the rain in guyana, for now at least — can england keep their t20 world cup defence going by beating india in their semi final. good evening. the bbc has tracked down and confronted a people smuggler responsible for organising a small—boat crossing to the uk in april in which a seven—year—old girl died. our paris correspondent andrew harding was on the beach in france when that boat set off. now after a lengthy investigation, he has managed to track down the smuggler across europe to luxembourg. here's his story. excuse me, sir. andrew harding from bbc news. we've come to luxembourg to confront a man we've been chasing across europe. we know who you are, you're a smuggler. no. he's responsible for putting hundreds of people in small boats heading from france to england. and responsible for one boat in particular. this one. we came across it back in april on a beach in northern france. police, battling with a group of smugglers. chaos a

>> it's an interference so if it's a bad gene, it blocks the gene. >> this is a disease, it's interestingr and it produces -- you have this protein that's misfolded and those proteins deposit in your heart or your nerves so that's why this same drug can work for both. >> it blocks the -- >> and then your heart can't pump and leads to heart failure. >> something wrong with one of your genes. >> there's hereditary and wild type. >> if it's rna interference one a way or another your dna is bad so you want to suppress the bad. alnylam has proven this technology works with other drugs. >> they have a few on the market -- >> this is not a proof of technology? >> no. >> >> it's already proven. it's actual market size for how many people have this, so it could be profitable for them? >> exactly. they have other drugs but those are smaller indications. this drug is approved for the nerve condition that is associated with this disease but that's a much smaller market so for them the company did about 1.2 billion in revenue last year. so they're saying this is the thing to make it bigger. i saw anal

how to preserve the gene pool of farm animals? it will not solve the problem of increasing productivity. that is, our current task is to create what is needed to create ideal optics that will help bring space closer. a highly qualified optician performs the manufacturing. surface, after his work the surface deviates from the ideal shape by a maximum of two to three microns. how does the weather in space affect the weather at home? a major solar event can cause damage and disruption in virtually every industry. at the end of the last century, one lamb was born, outwardly it was no different from its brothers, even the medal. identified 63. a little later the nickname was changed, so the world learned about that very little sheep, the first genetic copy of a living organism. since then, scientists have been able to clone many animals, from mice to macaques and camels. the place where i am now is also significant in this regard. the first cloned mammal in russia, the famous flower cow, appeared here. from the outside it all looks lik

, that is, yes, mutations in this gene can be detected not only in children with this disease, but in it’s impossible to say reliably why anna didn’t tell her mother-in-law about her daughter’s illness until recently; at home she cried at midnight. pillow, watch after a short pause, the life of director dmitry ostrakhan, how to the cinema. the main director has no right to sleep with an artist, what did he share with leonid germolnik? it wasn't like he was being capricious. leonid is an emotional person. dmitry khananovich shouted, right? i don't remember in what words. why is he hiding his first marriage? masha, you buried me early, when i left, i said a phrase about how he lost his wife and doctors during a fire. they said that she could not be saved, that she had a big burn and was ready to break the law for the sake of her son, of course, it was a severe injury, where dmitry ostrakhan found the love of all his life to say, yes or very good, it’s very easy to live with him, and how his ballerina wife saved the whole family during a terrible robbery, we are worried and you are worri

in boys, the chromosome set is 46 x,y, in girls it is 46x. and if a boy from... due to a broken gene,he muscles gradually stop working, but the insidiousness of the diagnosis is that at first it is imperceptible, until the age of 3-4 the child can look absolutely healthy, manifests itself in colossal weakness, he just getting up, sitting down, and some basic actions are quite difficult for us... it’s hard to imagine it. at this time , doctors are already preparing for the procedure. the drug was delivered to the russian children's clinical hospital in frozen form. temperature during transportation is strictly below 60°. these ampoules are almost like a magic medicine (elividis). this is what the drug looks like: the drug is individually calculated based on the child’s weight and prepared directly, specifically for each patient. one dropper is enough, and the disease that used to be considered incurable, will begin to recede. with the help of the elividis drug, it became possible to introduce the missing gene using an adenoviral vector and this gene appears in a sick child and... the b

an essential gene was removed from this taxi virus and replaced with an influenza virus gene. it initiates production of an avian flu protein in the muscle cells of vaccinated birds. a warning for the immune system, which then response by generating large numbers of anti bodies. a couple of the to get into the body reaction by producing and keep on agent. that's actually neutralizing i just saw units here which can neutralize the h 5 and one virus income. so this system has another advantage to attendance stimuli to simulate and unity blood samples show that all 24 of the captive bird species reacted penguins produced a particularly large number of anti bodies and initial infection trials with age 5 . then one in chickens also indicate a protective immune response. not a single chicken and the trials became mill. but what about the wild, migratory birds that spread the virus, or the endangered emperor or penguins, way down in the antarctic? in theory, the explanation could protect them to expert se, but distributing vaccines adequately and reliably to those populations remains

its expenditure, it is inherent actually in the genetic material, and there is a whole cascade of genesif certain disorders exist, do not store energy, or store it in excess, it is important under what conditions, but this genetic information is realized, so we now live in conditions of excess energy production, our entire culture, all our achievements of civilization, they are aimed at storing energy, but not spend, and cars, yes, on transport from from the cabin from the bed from your own... in the morning to the office and back, practically, if we don’t go to the fitness club every day, don’t swim in the pool, crawl or butterfly, then we practically are unable to spend it the amount of energy that we consume in these large portions that we now eat, mainly carbohydrate foods, it is absolutely true that obesity has always been considered an attribute - average and lower on the social ladder of this status, but now... people really have the opportunity to plan your life more harmoniously, spend energy, play sports, eat not fast food, in gourmet restaurants, remember what a gourmet dish

francisco lopera no ha dejado trabajar en el caso y ha encontrado dos genes que retrasa el inicio delstos dÍas, la comunidad cientÍfica se lo reconociÓ. [habla en inglÉs] reportero: a sus 73 aÑos, el cientÍfico colombiano recibiÓ un premio. >> entre los investigadores de enfermedades neurodegenerativa lo reconocen. reportero: esto podrÍa ser un premio de toda la vida, pero Él estÁ lejos de su retiro y cerca de una cura. >> hemos detectado dos genes que puede retrasar 20 aÑos el ciclo de la enfermedad. eso es muy esperanzador. significa que hay maneras de retrasar la enfermedad. reportero: segÚn la organizaciÓn mundial de la salud (oms), 55,000,000 de personas en el mundo padecen esta enfermedad. >> cuando comencÉ a estudiar medicina, hace mÁs de 40 aÑos, mi abuela tuvo esta enfermedad. reportero: segÚn Él puede tardar mÁs de cinco aÑos prevenir o curar la enfermedad. en mÉxico tambiÉn hay mutaciones de enfermedades de alzheimer al igual que en argentina, brasil, y otros paÍses mÁs. fÉlix: si les gustan las piscinas los vamos a llevar a conocer esta piscina en dubÁi. es la

that means, regarding the possible loss of some elements of european culture and, say, culture, or geneseuropean culture in russia due to the fact that we are somehow turning towards the east. only persist, but accelerate, intensify, bearing in mind the rate of economic growth today in the near future, this is an inevitable, inevitable course of events, no one can do anything about it, that’s what would n’t happen, this trend will continue, and of course... what concerns the elements of european culture, we are not losing it, we are not losing them, these elements and genes of european culture, in that part of europe called western, where is this european culture, it will soon not remain there at all, only architectural monuments, but culture is , first of all, the consciousness of people, it is poisoned by today's global liberalists, who... who, if you look a little deeper into the centuries, are based even for non-believers, in general, based, of course, primarily on christian culture, for us , for russia, for a multi-confessional country, you said, i don’t know who the author of these

using an adenoviral vector and this gene appears in a sick child, and the protein that is responsiblee begins to work fully, such an incredible effect and costs, accordingly, 260 million rubles. or the most expensive medicine in the world, 2 seconds more expensive than the already well-known zalgensma, which has become a salvation for children with spinal muscular atrophy. russia is one of the first countries in which the drug became available and available free of charge, thanks to state foundation circle of good. its budget is formed due to an increased tax rate for those whose annual income is more than 5 million rubles. this is a story without analogues in the world with the support of the president of the russian federation, the ministry of health of russia, drugs at the borders for 6 years. arsen will turn six in a week, and this is the best gift not only for his birthday, but for the rest of his life. at the last moment, when they announced such an opportunity, it, of course, stunned me, turned everything upside down in my head, it can’t be called anything other than a miracle.

i feel gratitude to the parents who gave me genes, well, because the genes are really good, the fatherisciplined my mind, gave me a clear understanding of childhood itself, what a goal is. in st. petersburg, football day was also celebrated on a grand scale. the festival consisted of more than 50 venues, and the main events took place at the kirovets stadium. the children's tournament was opened by zenit captain alexey igonin, then he was joined by former national team forward pavel pogrebnyak. the russian super cup was set up near the grounds where young football players competed, and adult teams played on the main field. the most titled club in the world, real madrid, has once again proven its greatness. at the legendary wembley cream beat borussia 2:0 in the champions league final. and for most of the meeting , dortmund played no worse, and maybe even better than their opponents. so in the first half, real had no shots on target, their goal was saved time after time by thibaut courtois. the same courtois who was not taken to the euro by ex-coach of spartak domenic tedesca. and in th

, which you the selfish gene, which you mentioned earlier, and it doesn't contradict the selfish genein respects. assuming that darwin's theory of natural selection is correct , it the selection is correct, it the thesis is that every animal, every living creature is a book which can be read as a description of the of the series of ancient worlds in which its ancestors survived. it's not possible at the moment, but i'm predicting that the zoologists of the future will be able to take any animal and read it as a story about its ancestral past and the idea that there isn't a sort of higher power, even if god doesn't exist , isn't god doesn't exist, isn't believing in him or her good. >> if it improves your quality of life, i suppose you could say that even if it's false, i personally believe in truth so much that i wouldn't regard that as a very good argument. >> i wouldn't regard simply improving your quality of life as being a good enough reason to believe in something that isn't true. if you think it is true, that's fine. but but, but you've put the question in the form of even if it